Enhanced uptake of antisense oligonucleotides using cationic liposomes and the apoptotic effect of idarubicin in K-562 cell line

Leuk Res. 2002 Jul;26(7):669-76. doi: 10.1016/s0145-2126(01)00191-6.

Abstract

We have evaluated the apoptotic and DNA damaging activity of Idarubicin (IDA) on K-562 cells alone and following the uptake of modified antisense-oligodeoxynucleotides (AS-ODNs) targeting b3a2 mRNA of bcr/abl hybrid gene, after treatment with AS-ODNs/DCChol-DOPE (liposomes) complexes. The uptake of FITC-labeled oligonucleotide-liposomes complexes (FITC-ODNs/DCChol-DOPE) was analyzed by flow cytometry and fluorescence microscopy. Both techniques indicated cytoplasmic accumulation of labeled liposome complexes following 24h of exposure. In absence of liposomes, AS-ODNs uptake was minimal. Pre-treatment of cells with AS-ODNs/DCChol-DOPE increased the capability of IDA to induce apoptosis as determined by morphology and the comet assay. In contrast, the use of a non-sense oligodeoxynucleotide conjugated with liposomes, in the presence of IDA, did not increase K-562 cell apoptosis. Nevertheless, DNA damage in IDA treated cells was not related to ODNs/liposomes pre-treatment, as determined by the comet assay. Our data suggests that DCChol-DOPE increases the uptake of ODNs in K-562 cells, and these modified AS-ODNs increase IDA induced apoptosis by decreasing p210(bcr/abl) levels in K-562 cells.

MeSH terms

  • Apoptosis / drug effects*
  • Biological Transport
  • Cations / chemistry
  • Cholesterol / analogs & derivatives*
  • Cholesterol / chemistry
  • Comet Assay
  • DNA Damage
  • DNA, Neoplasm / analysis
  • Drug Synergism
  • Flow Cytometry
  • Fusion Proteins, bcr-abl / biosynthesis
  • Fusion Proteins, bcr-abl / genetics
  • Gene Expression Regulation, Leukemic / drug effects
  • Humans
  • Idarubicin / pharmacology*
  • K562 Cells / drug effects*
  • K562 Cells / metabolism
  • Lipids / chemistry
  • Liposomes / administration & dosage
  • Liposomes / chemistry
  • Microscopy, Fluorescence
  • Neoplasm Proteins / biosynthesis
  • Neoplasm Proteins / genetics
  • Oligodeoxyribonucleotides, Antisense / administration & dosage
  • Oligodeoxyribonucleotides, Antisense / metabolism*
  • Oligodeoxyribonucleotides, Antisense / pharmacology
  • Phosphatidylethanolamines / chemistry
  • Quaternary Ammonium Compounds / chemistry
  • RNA, Messenger / antagonists & inhibitors
  • RNA, Messenger / genetics
  • RNA, Neoplasm / antagonists & inhibitors
  • RNA, Neoplasm / genetics

Substances

  • Cations
  • DNA, Neoplasm
  • Lipids
  • Liposomes
  • Neoplasm Proteins
  • Oligodeoxyribonucleotides, Antisense
  • Phosphatidylethanolamines
  • Quaternary Ammonium Compounds
  • RNA, Messenger
  • RNA, Neoplasm
  • 3-(N-(N',N'-dimethylaminoethane)carbamoyl)cholesterol
  • (3-dimyristyloxypropyl)(dimethyl)(hydroxyethyl)ammonium
  • dioleoyl phosphatidylethanolamine
  • Cholesterol
  • Fusion Proteins, bcr-abl
  • Idarubicin