Very early-onset familial Alzheimer's disease: a novel presenilin 1 mutation

Int J Geriatr Psychiatry. 2002 Jul;17(7):649-51. doi: 10.1002/gps.657.

Abstract

Background: Early-onset familial Alzheimer's disease (EOFAD) is linked to mutations in three autosomal dominant genes: PS1, PS2 and APP. The clinical presentation and age of onset of mutations is variable.

Objectives: The aim of this report is to describe a novel PS1 mutation believed to be causal for a very early onset of AD.

Methods: This is a case history using information from medical records, relative interviews and genetic testing results to describe the pre-clinical prodrome and clinical course of a patient with EOFAD.

Results: A previously undescribed G206V mutation in PS1 was found in the proband.

Conclusion: The G206V mutation in PS1 is probably causal of a case of EOFAD with significant premorbid features.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Age of Onset
  • Alzheimer Disease / genetics*
  • Genetic Predisposition to Disease / genetics*
  • Humans
  • Male
  • Membrane Proteins / genetics*
  • Mutation*
  • Pedigree
  • Presenilin-1

Substances

  • Membrane Proteins
  • PSEN1 protein, human
  • Presenilin-1