Long-chain 3-hydroxyacylCoA dehydrogenase deficiency: a new case presenting with liver dysfunction, cholestasis and fibrosis

Acta Paediatr. 2002;91(6):719-22. doi: 10.1080/080352502760069197.

Abstract

A cholestatic 6-mo-old girl was admitted to our department because she recently presented with hypotonia and lethargy, apparently due to moderate and transient hypoglycaemia. Her urine contained 3-hydroxy-dicarboxylic acids of 12 to 14 carbons in length and her plasma acylcarnitine profile was consistent with long-chain 3-hydroxyacylCoA dehydrogenase deficiency. This diagnosis was confirmed by enzyme studies. This deficiency was due to a G1528C mutation on the paternal allele (mutation on the maternal allele as yet not identified). The patient improved dramatically with medium-chain triglyceride supplementation.

Conclusion: Early cholestasis and hepatic fibrosis must lead to search for long-chain 3-hydroxyacylCoA dehydrogenase deficiency, particularly when hypoketotic hypoglycaemia is present.

Publication types

  • Case Reports

MeSH terms

  • Acyl-CoA Dehydrogenase, Long-Chain
  • Biopsy, Needle
  • Cholestasis / etiology*
  • Cholestasis / pathology
  • Fatty Acid Desaturases / deficiency*
  • Fatty Acid Desaturases / metabolism*
  • Female
  • Follow-Up Studies
  • Humans
  • Infant
  • Lipid Metabolism, Inborn Errors / complications*
  • Lipid Metabolism, Inborn Errors / diagnosis*
  • Lipid Metabolism, Inborn Errors / diet therapy
  • Liver Cirrhosis / etiology*
  • Liver Cirrhosis / pathology
  • Risk Assessment
  • Severity of Illness Index

Substances

  • Fatty Acid Desaturases
  • Acyl-CoA Dehydrogenase, Long-Chain