Abstract
Chronic graft-versus-host disease (GVHD) accompanying autoimmune disease was induced in (C57BL/6xDBA/2) F(1) mice (H-2(b/d)) by an injection of splenic T cells of parental DBA/2 origin (H-2(d)). In parallel with the onset of proteinuria, an expansion of lymphocytes was induced in the liver and kidney, showing a peak at 2 weeks after the onset of disease. The majority of lymphocytes were of recipient origin (H-2(b/d)). The main lymphocyte subset among T cells at the pre-onset stage and after the onset of disease was CD8(+) NK1.1(-) CD3(int) cells (of extrathymic, hepatic origin) in both the liver and kidney. NK1.1(-) CD3(int) cells confer primarily neither NK-like nor NKT-like cytotoxicity. No induction of these types of cytotoxicity was observed in these mice with the expansion of NK1.1(-) CD3(int) cells. This raised the possibility that granulocytes induced in the liver and kidney might be associated with tissue damage. The present results suggest that, similarly to the case of autoimmune-prone mice with genetic background (e.g. MRL-lpr/lpr mice and BXSB mice), NK1.1(-) CD3(int) cells of extrathymic, hepatic origin might be crucial lymphocytes involved in the induction of the autoimmune-like disease in mice with chronic GVHD, in conjunction with Bcells (e.g. B-1 cells).
MeSH terms
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Adoptive Transfer
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Animals
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Antigens / analysis*
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Antigens, Ly
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Antigens, Surface
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Autoantibodies / biosynthesis
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Autoantibodies / immunology
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Autoimmune Diseases / etiology
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Autoimmune Diseases / immunology*
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Autoimmune Diseases / pathology
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B-Lymphocyte Subsets / immunology
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CD3 Complex / analysis
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CD4-CD8 Ratio
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CD5 Antigens / analysis
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CD8-Positive T-Lymphocytes / chemistry
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CD8-Positive T-Lymphocytes / immunology
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CD8-Positive T-Lymphocytes / transplantation
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Chronic Disease
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Crosses, Genetic
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Cytotoxicity, Immunologic
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Female
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Graft vs Host Disease / etiology
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Graft vs Host Disease / immunology*
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Granulocytes / immunology
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Granulocytes / pathology
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H-2 Antigens / immunology
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Immunoglobulin D / biosynthesis
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Immunoglobulin D / immunology
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Immunoglobulin M / biosynthesis
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Immunoglobulin M / immunology
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Kidney / immunology
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Kidney / pathology
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Killer Cells, Natural / immunology*
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Lectins, C-Type
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Liver / immunology
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Liver / pathology
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Lymphocyte Activation
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Lymphoproliferative Disorders / etiology
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Lymphoproliferative Disorders / immunology*
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Mice
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Mice, Inbred C57BL
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Mice, Inbred DBA
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Mice, Inbred MRL lpr
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Models, Animal
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NK Cell Lectin-Like Receptor Subfamily B
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Nephritis / etiology
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Nephritis / immunology*
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Nephritis / pathology
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Proteins / analysis*
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Proteinuria / etiology
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Specific Pathogen-Free Organisms
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Spleen / immunology
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T-Lymphocyte Subsets / chemistry
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T-Lymphocyte Subsets / immunology
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T-Lymphocyte Subsets / transplantation*
Substances
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Antigens
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Antigens, Ly
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Antigens, Surface
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Autoantibodies
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CD3 Complex
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CD5 Antigens
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H-2 Antigens
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Immunoglobulin D
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Immunoglobulin M
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Klrb1c protein, mouse
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Lectins, C-Type
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NK Cell Lectin-Like Receptor Subfamily B
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Proteins