Arginine metabolism in Trypanosoma cruzi is coupled to parasite stage and replication

FEBS Lett. 2002 Aug 28;526(1-3):111-4. doi: 10.1016/s0014-5793(02)03157-5.

Abstract

L-Arginine plays an essential role in the energetic metabolism of Trypanosoma cruzi. In this work we propose a relationship between L-arginine uptake, arginine kinase activity and the parasite replication ability. In epimastigote cultures L-arginine uptake decreases continuously accompanying a cell replication rate reduction. The use of conditioned or fresh medium mimics uptake variations. Interestingly, in non-replicative trypomastigote cells, L-arginine uptake was undetectable. The association between L-arginine uptake and cell replication was demonstrated using the antimitotic agent hydroxyurea. Arginine kinase, the enzyme responsible for phosphoarginine and ATP synthesis, also shows a differential activity in epimastigote and trypomastigote parasite stages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Arginine / metabolism*
  • Arginine Kinase / metabolism
  • Biological Transport
  • Life Cycle Stages
  • Trypanosoma cruzi / growth & development*
  • Trypanosoma cruzi / metabolism

Substances

  • Arginine
  • Arginine Kinase