Stimulation of growth-hormone release with clonidine does not distinguish individual cases of idiopathic Parkinson's disease from those with striatonigral degeneration

J Neurol. 2002 Sep;249(9):1206-10. doi: 10.1007/s00415-002-0809-y.

Abstract

Multiple System Atrophy (MSA) and idiopathic Parkinson's disease (PD) can be difficult to distinguish. There is an ongoing debate about the diagnostic value of the growth-hormone response to clonidine (CGH-test) in PD and MSA. We investigated whether the CGH-test can identify individual patients in the early stages of PD (n = 21) and Striatonigral Degeneration (SND, n = 11), a particular variety of MSA. Patients were diagnosed on the basis of clinical criteria and IBZM-SPECT. Clonidine induced a greater total serum growth-hormone production in PD than in SND (p = 0.01). However, taking the difference in prevalence of PD and SND into account, and because of the low likelihood ratios of the test, an increase of GH after clonidine increases the pre-test probability for PD by about only 5 %, while an absent response of GH also increases the pre-test probability for SND by about 5 %. We conclude that the CGH-test discriminates between groups of patients with PD and SND, but has little practical diagnostic value for identifying individual patients.

Publication types

  • Comparative Study

MeSH terms

  • Aged
  • Analysis of Variance
  • Chi-Square Distribution
  • Clonidine*
  • Diagnosis, Differential
  • Growth Hormone / blood
  • Growth Hormone / metabolism*
  • Humans
  • Middle Aged
  • Parkinson Disease / blood
  • Parkinson Disease / diagnosis*
  • Statistics, Nonparametric
  • Striatonigral Degeneration / blood
  • Striatonigral Degeneration / diagnosis*

Substances

  • Growth Hormone
  • Clonidine