Genetic sources of covariation among P3(00) and online performance variables in a delayed-response working memory task

Biol Psychol. 2002 Oct;61(1-2):183-202. doi: 10.1016/s0301-0511(02)00058-3.

Abstract

Genetic and environmental sources of covariation among the P3(00) and online performance elicited in a delayed-response working memory task, and psychometric IQ assessed by the multidimensional aptitude battery, were examined in an adolescent twin sample. An association between frontal P3 latency and task performance (phenotypic r=-0.33; genotypic r=-0.49) was indicated, with genes (i.e. twin status) accounting for a large part of the covariation (>70%). In contrast, genes influencing P3 amplitude mediated only a small part (2%) of the total genetic variation in task performance. While task performance mediated 15% of the total genetic variation in IQ (phenotypic r=0.22; genotypic r=0.39) there was no association between P3 latency and IQ or P3 amplitude with IQ. The findings provide some insight into the inter-relationships among psychophysiological, performance and psychometric measures of cognitive ability, and provide support for a levels-of-processing genetic model of cognition where genes act on specific sub-components of cognitive processes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Twin Study

MeSH terms

  • Adolescent
  • Adult
  • Analysis of Variance
  • Aptitude Tests
  • Arousal / physiology*
  • Cerebral Cortex / physiology
  • Event-Related Potentials, P300 / genetics*
  • Female
  • Genotype
  • Humans
  • Individuality
  • Intelligence / genetics*
  • Male
  • Mental Recall / physiology*
  • Orientation / physiology
  • Pattern Recognition, Visual / physiology
  • Phenotype
  • Psychomotor Performance / physiology
  • Reaction Time / genetics*
  • Twins, Dizygotic / genetics*
  • Twins, Monozygotic / genetics*