[Expression and significance of bcl-2 and bcl-xL in rat neuron after acute brain trauma]

Zhonghua Wai Ke Za Zhi. 2002 Sep;40(9):702-4.
[Article in Chinese]

Abstract

Objective: To observe the expression of bcl-2 and bcl-xL in rat brain neuron after acute brain trauma.

Methods: The rat model of mild and sever brain trauma were made by diffused brain injury. The expression of bcl-2 and bcl-xL in rat brain neuron was examined by immunohistochemical staining. Rat neuron apoptosis was detected by TUNEL method.

Results: There were few bcl-2 (cortex: 4.40 +/- 1.67, hippocampal: 3.20 +/- 1.30) and lots of bcl-xL (cortex: 45.60 +/- 4.34, hippocampal: 50.20 +/- 3.50) expression in normal controls. After impact, the expression of bcl-2 in rat brain neuron increased, most distinctly on day 1(cortex: 30.0 +/- 4.3, hippocampal: 46.6 +/- 3.2), in mild group. A negative correlation was seen between bcl-2 expression and neuronal apoptosis (-1 < r < -0.847, P< 0.01, n = 10). No change was seen in bcl-xL expression.

Conclusions: bcl-2 expression increased after acute brain trauma but bcl-xL did not change much. Both bcl-2 and bcl-xL are concerned with anti-apoptosis in neuron after acute brain trauma.

Publication types

  • English Abstract

MeSH terms

  • Acute Disease
  • Animals
  • Apoptosis
  • Brain Injuries / metabolism*
  • Brain Injuries / pathology
  • Immunohistochemistry
  • Male
  • Neurons / chemistry*
  • Neurons / pathology
  • Proto-Oncogene Proteins c-bcl-2 / analysis*
  • Rats
  • Rats, Wistar
  • bcl-X Protein

Substances

  • Bcl2l1 protein, rat
  • Proto-Oncogene Proteins c-bcl-2
  • bcl-X Protein