Host genetic profiles predict virological and immunological control of HIV-1 infection in adolescents

AIDS. 2002 Nov 22;16(17):2275-84. doi: 10.1097/00002030-200211220-00007.

Abstract

Objective: To evaluate the correlation between host genetic profiles and virological and immunological outcomes among HIV-1-seropositive participants from the Reaching for Excellence in Adolescent Care and Health (REACH) cohort.

Methods: HLA class I and chemokine coreceptor (CCR) alleles and haplotypes were resolved in 227 HIV-1-seropositive adolescents (ages 13-18 years; 75% females; 71% African-Americans) and 183 HIV-seronegative individuals, with quarterly follow-up visits between 1996 and 2000. Each HLA and CCR variant with consistent risk and protective effect on HIV-1 pathogenesis was assigned a score of -1 and +1, respectively. All individual markers and genetic scores were analyzed in relation to plasma viral load (VL) and CD4 T lymphocytes during a 6-12-month interval when no antiretroviral therapy was taken.

Results: HLA-B*57 alone was a strong predictor of VL (P < 0.0001), but composite genetic profiles found in over 50% of patients consistently outperformed the individual component markers in multivariable analyses with or without adjustment for gender, race, age, and membership of clinical patient groups. Adolescents (n = 37) with a favorable combination of VL (< 1000 copies/ml) and CD4 T cell counts (> 450 x 10(6) cells/l) consistently had more positive (+1 to +2) than negative (-1 to -4) HLA and CCR scores compared with those (n = 56) with an unfavorable combination (VL > 16,000 copies/ml and CD4 cells < 450 x 10(6) cells/l) or the remainder (n = 134) of the cohort (overall P < 0.0001).

Conclusion: A generalizable genetic scoring algorithm based on seven HLA class I and CCR markers is highly predictive of viremia and immunodeficiency in HIV-1-infected adolescents.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Algorithms
  • Alleles
  • CD4 Lymphocyte Count
  • Female
  • Follow-Up Studies
  • Genes, MHC Class II*
  • Genetic Markers
  • Genetic Predisposition to Disease*
  • HIV Infections / genetics*
  • HIV Infections / immunology
  • HIV Infections / virology
  • HIV-1*
  • Haplotypes
  • Histocompatibility Testing
  • Humans
  • Male
  • Prognosis
  • Receptors, CCR2
  • Receptors, Chemokine / genetics*
  • Viral Load
  • Viremia / genetics
  • Viremia / immunology
  • Viremia / virology

Substances

  • CCR2 protein, human
  • Genetic Markers
  • Receptors, CCR2
  • Receptors, Chemokine