Antibodies against GluR3 peptides are not specific for Rasmussen's encephalitis but are also present in epilepsy patients with severe, early onset disease and intractable seizures

J Neuroimmunol. 2002 Oct;131(1-2):179-85. doi: 10.1016/s0165-5728(02)00261-8.

Abstract

Rasmussen's encephalitis (RE) is a rare condition characterized by drug-resistant seizures, recurrent status epilepticus and progressive lateralized neurological deterioration. There is evidence of autoimmune involvement in the pathogenesis. We investigated the presence of anti-GluR3 antibodies against peptides A and B in patients with RE (n=11), partial and generalized epilepsy (n=85) and other neurological diseases (n=30). The antibodies were specific for epilepsy and are thus not a marker of RE, while particularly high antibody titers characterized a subgroup of non-RE patients with "catastrophic" epilepsy. Antibodies against GluR3B peptide were significantly associated with frequent seizures compared to occasional or drug-controlled seizures.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Age of Onset
  • Antibody Specificity
  • Autoantibodies / cerebrospinal fluid
  • Autoantibodies / immunology*
  • Autoimmune Diseases of the Nervous System / immunology
  • Child
  • Child, Preschool
  • Encephalitis / immunology*
  • Epilepsy / diagnosis
  • Epilepsy / immunology*
  • Epitopes / immunology
  • Female
  • Humans
  • Infant
  • Male
  • Nervous System Diseases / immunology
  • Peptides / immunology
  • Receptors, AMPA
  • Receptors, Glutamate / chemistry
  • Receptors, Glutamate / immunology*

Substances

  • Autoantibodies
  • Epitopes
  • Peptides
  • Receptors, AMPA
  • Receptors, Glutamate
  • glutamate receptor ionotropic, AMPA 3