Effect of Korea red ginseng on cerebral blood flow and superoxide production

Acta Pharmacol Sin. 2002 Dec;23(12):1152-6.

Abstract

Aim: To investigate the effects of Korea red ginseng (KRG) on the cerebral perfusion rate in the rats and the generation of superoxide anion in the endothelial cells.

Methods: The cerebral perfusion rate was measured using laser-doppler flowmetry before and after the administration of crude saponin (CS) and saponin-free fraction (SFF) of KRG in the anesthetized rats. The superoxide generation was measured by the method based on lucigenin-enhanced chemiluminescence in the cultured endothelial cells.

Results: The relative cerebral perfusion rate (rCBF) was significantly increased by the intraperitoneal injection of CS (100 mg/kg) in the rats, but SFF had no effect on the rCBF. Chronic treatment with CS for 7 d significantly inhibited the decrease of forebrain cerebral blood flow induced by clamping both carotid arteries in the rats. Furthermore, CS (0.1 g/L) significantly suppressed NADPH-induced superoxide generation in the human umbilical vein endothelial cells (P <0.01).

Conclusion: The present study demonstrated that crude saponin fraction of KRG enhanced cerebral blood flow in rats. Furthermore, crude saponin fraction of KRG abrogated the NADPH-driven superoxide generation in endothelial cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • Cerebral Cortex / blood supply
  • Cerebral Cortex / drug effects*
  • Cerebrovascular Circulation / drug effects*
  • Endothelial Cells / metabolism
  • Endothelium, Vascular / cytology
  • Endothelium, Vascular / metabolism
  • Ginsenosides / isolation & purification
  • Ginsenosides / pharmacology*
  • Humans
  • Neuroprotective Agents / isolation & purification
  • Neuroprotective Agents / pharmacology*
  • Panax / chemistry*
  • Plant Roots / chemistry
  • Plants, Medicinal / chemistry
  • Rats
  • Superoxides / metabolism*
  • Umbilical Veins / cytology
  • Umbilical Veins / metabolism

Substances

  • Ginsenosides
  • Neuroprotective Agents
  • Superoxides