Calcium-dependent conformational changes of membrane-bound Ebola fusion peptide drive vesicle fusion

FEBS Lett. 2003 Jan 30;535(1-3):23-8. doi: 10.1016/s0014-5793(02)03847-4.

Abstract

The fusogenic subdomain of the Ebola virus envelope glycoprotein is an internal sequence located ca. 20 residues downstream the N-terminus of the glycoprotein transmembrane subunit. Partitioning of the Ebola fusion peptide into membranes containing phosphatidylinositol in the absence of Ca2+ stabilizes an alpha-helical conformation, and gives rise to vesicle efflux but not vesicle fusion. In the presence of millimolar Ca2+ the membrane-bound peptide adopts an extended beta-structure, and induces inter-vesicle mixing of lipids. The peptide conformational polymorphism may be related to the flexibility of the virus-cell intermembrane fusogenic complex.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Calcium / chemistry*
  • Circular Dichroism
  • Ebolavirus / chemistry*
  • Lipids / chemistry
  • Liposomes / chemistry
  • Membrane Fusion / physiology*
  • Membranes, Artificial
  • Peptide Fragments / chemistry*
  • Peptide Fragments / metabolism
  • Phosphatidylinositols / chemistry
  • Protein Conformation
  • Protein Structure, Secondary
  • Spectrophotometry, Infrared
  • Viral Fusion Proteins / chemistry*
  • Water / chemistry

Substances

  • Lipids
  • Liposomes
  • Membranes, Artificial
  • Peptide Fragments
  • Phosphatidylinositols
  • Viral Fusion Proteins
  • Water
  • Calcium