GJB2 deafness gene shows a specific spectrum of mutations in Japan, including a frequent founder mutation

Hum Genet. 2003 Apr;112(4):329-33. doi: 10.1007/s00439-002-0889-x. Epub 2003 Jan 31.

Abstract

Mutations in the GJB2 gene (connexin 26) are the major cause of autosomal recessive non-syndromic hearing impairment in many populations. In contrast to the volume of information regarding the involvement of GJB2 mutations in hearing impairment in populations of European ancestry, less is known regarding other ethnic groups. In this study, we analyzed the GJB2 gene for mutations in 1227 hearing-impaired Japanese individuals. This revealed a unique spectrum of GJB2 mutations, different from that found in the Caucasian population. The most frequent mutation in Japanese, 235delC, has never been reported in Caucasians. To investigate a possible founder effect for the 235delC mutation, we analyzed single nucleotide polymorphisms in the vicinity of the GJB2 gene. Results were consistent with inheritance of the 235delC mutation from a common ancestor. The results of this study have important implications for genetic diagnostic testing for deafness in the Japanese population.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Asian People / genetics*
  • Case-Control Studies
  • Connexin 26
  • Connexins / genetics*
  • Deafness / genetics
  • Founder Effect*
  • Hearing Loss, Sensorineural / genetics*
  • Heterozygote
  • Homozygote
  • Humans
  • Japan
  • Molecular Sequence Data
  • Mutation*
  • Polymorphism, Single Nucleotide / genetics*

Substances

  • Connexins
  • GJB2 protein, human
  • Connexin 26

Associated data

  • GENBANK/M86849