[Regulation of Gal beta 1,3GalNAc alpha 2,3-sialyltransferase (ST3GalI) by hepatitis B virus MHBst/HBx transactivator]

Sheng Wu Gong Cheng Xue Bao. 2002 Sep;18(5):551-5.
[Article in Chinese]

Abstract

Hepatitis B virus MHBst and HBx fragments were amplified to construct eukaryotic expression vector pCDNA3.1-MHBst and pCDNA3.1-HBx. ST3GalI promoter region was obtained by the method of PCR and GFP report plasmid pEGFP-N1-Psial was constructed. pCDNA3.1-MHBst or pCDNA3.1-HBx with pEGFP-N1-Psial were transiently co-transfected into QGY-7701 cells using calcium phosphate-DNA co-precipitation, respectively. The expressions of Psial-directed GFP were analyzed by FAC-Scalibur. It was found that MHBst/HBx could upregulate ST3GalI promoter activity by 35.2% and 43.8%, respectively. We report the regulation of ST3GalI by MHBst and HBx transactivators. It would be helpful to further investigate the relation between hepatitis B virus infection and sialyltransferase expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Gene Expression Regulation, Enzymologic*
  • Hepatitis B Surface Antigens / genetics
  • Hepatitis B Surface Antigens / physiology*
  • Hepatitis B virus
  • Promoter Regions, Genetic
  • Sialyltransferases / genetics*
  • Trans-Activators / genetics
  • Trans-Activators / physiology*
  • Transfection
  • Viral Regulatory and Accessory Proteins

Substances

  • Hepatitis B Surface Antigens
  • Trans-Activators
  • Viral Regulatory and Accessory Proteins
  • hepatitis B virus X protein
  • Sialyltransferases
  • N-acetyllactosaminide alpha-2,3-sialyltransferase