Apoptosis, ageing and cancer susceptibility

Br J Cancer. 2003 Feb 24;88(4):487-90. doi: 10.1038/sj.bjc.6600767.

Abstract

We have previously shown that peripheral blood lymphocytes (PBL) from individuals carrying a germline TP53 mutation show a dramatically reduced apoptotic response to radiation. As part of a study of this phenomenon, we also investigated apoptotic response in a series of breast cancer patients lacking TP53 mutations and in a control group of individuals without cancer. There was a significant reduction in mean apoptotic response with increasing age in all groups. These findings are consistent with a number of studies in rodents, which have demonstrated a reduction in DNA damage-induced apoptosis with increasing age. In addition, after adjusting for age, breast cancer patients showed significantly reduced apoptotic responses compared with normal controls (P=0.002). The odds ratio for breast cancer in women with an apoptotic response of <35%, compared with women with a response of >49%, was 6.42 (95% CI 1.68-24.6). The data further support the hypothesis that a reduction in apoptotic response to DNA damage with increasing age may play a significant role in the age-related increase in cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aging / physiology*
  • Apoptosis* / radiation effects
  • BRCA1 Protein / genetics
  • BRCA2 Protein / genetics
  • Disease Susceptibility*
  • Dose-Response Relationship, Radiation
  • Female
  • Gamma Rays
  • Humans
  • Lymphocytes / pathology
  • Lymphocytes / radiation effects
  • Male
  • Middle Aged
  • Neoplasms / epidemiology
  • Neoplasms / genetics*
  • Neoplasms / pathology*
  • Odds Ratio
  • Sex Characteristics

Substances

  • BRCA1 Protein
  • BRCA2 Protein