Ultraviolet irradiation increases FADD protein in apoptotic human keratinocytes

Biochem Biophys Res Commun. 2003 Mar 7;302(2):290-5. doi: 10.1016/s0006-291x(03)00186-4.

Abstract

Ultraviolet irradiation (UV) can induce keratinocyte apoptosis by activating death receptors that recruit the intracellular adaptor molecule FADD/MORT1 (Fas-associating death domain protein/mediator of receptor-induced toxicity). We hypothesized that UV could alter FADD expression levels to augment UV-induced keratinocyte apoptosis. In a dose-dependent manner UV B irradiation increased the expression of FADD protein in a human keratinocyte cell line (CCD-1106) with a corresponding increase in caspase-8 cleavage and cellular apoptosis. FADD overexpression induced cell death in 80% of cells compared with 10% spontaneous cell death in controls. Inhibition of FADD protein by adenoviral expression of anti-sense FADD reduced keratinocyte apoptosis. Regulation of FADD expression by UV may serve to enhance death receptor-mediated keratinocyte death.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Apoptosis / physiology*
  • Arabidopsis Proteins*
  • Cell Survival / radiation effects
  • Cells, Cultured
  • Fatty Acid Desaturases / metabolism*
  • Humans
  • Keratinocytes / metabolism
  • Keratinocytes / radiation effects*
  • Ultraviolet Rays*

Substances

  • Arabidopsis Proteins
  • Fatty Acid Desaturases
  • Fad7 protein, Arabidopsis