Bystander CD4 T cells do not mediate demyelination in mice infected with a neurotropic coronavirus

J Neuroimmunol. 2003 Apr;137(1-2):42-50. doi: 10.1016/s0165-5728(03)00041-9.

Abstract

Demyelination following infection of mice with the neurotropic coronavirus mouse hepatitis virus strain JHM (MHV) is immune-mediated. It has been demonstrated that MHV-specific CD4 and CD8 T cells are capable of causing demyelination independent of the other T cell subset. Recent work has also demonstrated that activated bystander CD8 T cells mediate significant demyelination. The ability of bystander CD4 T cells to mediate demyelination was investigated using CD4 T cell transgenic mice. The results indicated that bystander CD4 T cells were unable to cause demyelination in MHV-infected mice, despite being recruited into the central nervous system (CNS) and irrespective of activation status. These results suggest that CD4 T cells must recognize antigen in the CNS in order to cause demyelination.

MeSH terms

  • Animals
  • Bystander Effect / immunology*
  • CD4-Positive T-Lymphocytes / immunology*
  • Coronavirus / immunology*
  • Coronavirus Infections / genetics
  • Coronavirus Infections / immunology*
  • Demyelinating Diseases / genetics
  • Demyelinating Diseases / immunology*
  • Genes, RAG-1 / genetics
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic