Acute P. falciparum malaria induces a loss of CD28- T IFN-gamma producing cells

Parasite Immunol. 2002 Nov-Dec;24(11-12):545-8. doi: 10.1046/j.1365-3024.2002.00599.x.

Abstract

P. falciparum malaria is associated with increased activation among peripheral lymphocytes. In the present study, we investigated markers of susceptibility to apoptosis and expression of IFN-gamma and IL-4 by CD28-and CD28+T cells in West African children with acute P. falciparum malaria. The study showed increased susceptibility to apoptosis and cytokine production among T lymphocytes during acute malaria but also that T cells, in particular IFN-gamma producing CD28-T cells, were substantially reduced. These results are in line with previous studies suggesting that certain T cell subsets are sequestered away from the peripheral blood during P. falciparum malaria.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Animals
  • Apoptosis
  • CD28 Antigens / analysis*
  • Genes, bcl-2
  • Humans
  • Interferon-gamma / biosynthesis*
  • Interleukin-4 / analysis
  • Malaria, Falciparum / immunology*
  • Plasmodium falciparum / pathogenicity
  • T-Lymphocyte Subsets / classification
  • T-Lymphocytes / immunology*
  • fas Receptor / analysis

Substances

  • CD28 Antigens
  • fas Receptor
  • Interleukin-4
  • Interferon-gamma