Hormonal contraception can suppress natural antimicrobial gene transcription in human endometrium

Fertil Steril. 2003 Apr;79(4):856-63. doi: 10.1016/s0015-0282(02)04930-0.

Abstract

Objective: To determine the effect of hormonal contraception with a combined oral contraceptive pill and levonorgestrel intrauterine system on the expression of the natural antimicrobials secretory leukocyte protease inhibitor, beta-defensins 1 and 2, and granulysin in human endometrium.

Design: Observational study.

Setting: Day case ward in a department of obstetrics and gynecology.

Patient(s): Fifty seven women undergoing gynecologic procedures for benign conditions; 24 received no contraception for more than 3 months, 20 received a combined oral contraceptive for more than 3 months, and 13 wore a levonorgestrel intrauterine system for more than 3 months.

Main outcome measure(s): Endometrial samples were collected from all women. Messenger RNA was extracted and quantitative polymerase chain reaction was used to investigate expression of secretory leukocyte protease inhibitor, beta-defensin 1, beta-defensin 2, and granulysin. Immunohistochemistry for secretory leukocyte protease inhibitor was performed.

Result(s): All antimicrobials varied cyclically. The level of secretory leukocyte protease inhibitor was maximal in the late secretory and menstrual phase, beta-defensin 1 in the mid secretory phase, granulysin in the late secretory phase, and beta-defensin 2 in the menstrual phase. Use of a combined oral contraceptive or levonorgestrel intrauterine system use decreased messenger RNA expression of beta-defensin 1 and 2 and granulysin but not secretory leukocyte protease inhibitor.

Conclusion(s): Endogenous and exogenous sex-steroid hormones, in the form of a combined oral contraceptive or levonorgestrel intrauterine system, influence gene transcription of secretory leukocyte protease inhibitor, beta-defensin 1, beta-defensin 2, and granulysin in the endometrium.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Differentiation, T-Lymphocyte / biosynthesis*
  • Antigens, Differentiation, T-Lymphocyte / genetics
  • Biopsy
  • Contraceptives, Oral, Combined / pharmacology*
  • Endometrium / drug effects*
  • Endometrium / metabolism*
  • Endometrium / physiology
  • Female
  • Gene Expression Regulation / drug effects
  • Humans
  • Immunohistochemistry
  • Intrauterine Devices
  • Levonorgestrel / administration & dosage*
  • Levonorgestrel / pharmacology
  • Menstrual Cycle / drug effects
  • Menstrual Cycle / metabolism
  • Protein Biosynthesis*
  • Proteinase Inhibitory Proteins, Secretory
  • Proteins / genetics
  • RNA / biosynthesis
  • RNA / genetics
  • Reverse Transcriptase Polymerase Chain Reaction
  • Transcription, Genetic / drug effects
  • beta-Defensins / biosynthesis*
  • beta-Defensins / genetics

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • Contraceptives, Oral, Combined
  • GNLY protein, human
  • Proteinase Inhibitory Proteins, Secretory
  • Proteins
  • beta-Defensins
  • Levonorgestrel
  • RNA