Alpha v beta 5 integrin-dependent programmed cell death triggered by a peptide mimic of annexin V

Mol Cell. 2003 May;11(5):1151-62. doi: 10.1016/s1097-2765(03)00138-2.

Abstract

The diverse cytoplasmic domain sequences within the various integrin subunits are critical for integrin-mediated signaling into the cell (outside-in signaling) and for activation of ligand binding affinity (inside-out signaling). Here we introduce an approach based on phage display technology to identify molecules that specifically interact with the cytoplasmic domain of the beta 5 integrin subunit. We show that a peptide selected for binding specifically to the beta 5 cytoplasmic domain (VVISYSMPD) induces apoptosis upon internalization. The cell death process induced by VVISYSMPD is sensitive to modulation by growth factors and by protein kinase C (PKC), and it cannot be triggered in beta 5 null cells. Finally, we show that the VVISYSMPD peptide is a mimic of annexin V. Our results suggest a functional link between the alpha v beta 5 integrin, annexin V, and programmed cell death. We propose the term "endothanatos" to designate this phenomenon.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence / genetics
  • Annexin A5 / metabolism*
  • Apoptosis / physiology*
  • Cell Adhesion / physiology*
  • Cell Line
  • Cell Membrane / metabolism*
  • Eukaryotic Cells / metabolism*
  • Genetic Vectors
  • Growth Substances / metabolism
  • Growth Substances / pharmacology
  • Humans
  • Integrins / metabolism*
  • Molecular Mimicry / physiology
  • Molecular Sequence Data
  • Peptides / genetics
  • Peptides / metabolism
  • Peptides / pharmacology
  • Protein Kinase C / antagonists & inhibitors
  • Protein Kinase C / metabolism
  • Protein Structure, Tertiary / physiology
  • Receptors, Vitronectin / metabolism*
  • Signal Transduction / physiology

Substances

  • Annexin A5
  • Growth Substances
  • Integrins
  • Peptides
  • Receptors, Vitronectin
  • integrin alphaVbeta5
  • Protein Kinase C