EEC syndrome type 3 with a heterozygous germline mutation in the P63 gene and B cell lymphoma

Am J Med Genet A. 2003 Jul 30;120A(3):370-3. doi: 10.1002/ajmg.a.20064.

Abstract

Lines of evidence have recently indicated a relationship between mutations in the P63 gene and ectrodactyly-ectodermal dysplasia-clefting (EEC) syndrome type 3 (EEC3). The p63 gene (P63) has homology to P53 known as a tumor-suppressor gene, but biological function of its protein has not yet been known well. There have been two reported patients who had EEC syndrome associated with malignant lymphoma. However, they did not undergo sequencing analysis of P63. Here, we present with a Japanese girl who had EEC3 and developed diffuse large B-cell type non-Hodgkin lymphoma. In this patient, we documented a heterozygous germline mutation, Asp312Gly, in P63. We speculated that p63 may exert a biological function as a tumor suppressor. Malignant lymphoma should be considered as an important complication of EEC3.

Publication types

  • Case Reports

MeSH terms

  • Adolescent
  • DNA-Binding Proteins
  • Ectodermal Dysplasia / genetics*
  • Ectodermal Dysplasia / physiopathology
  • Female
  • Genes, Tumor Suppressor
  • Heterozygote
  • Humans
  • Japan
  • Lymph Nodes / pathology
  • Lymphoma, B-Cell / genetics*
  • Phosphoproteins / genetics*
  • Syndrome
  • Trans-Activators / genetics*
  • Transcription Factors
  • Tumor Suppressor Proteins

Substances

  • DNA-Binding Proteins
  • Phosphoproteins
  • TP63 protein, human
  • Trans-Activators
  • Transcription Factors
  • Tumor Suppressor Proteins