Mutation in mitochondrial tRNA(Leu)(UUR) gene in a large pedigree with maternally transmitted type II diabetes mellitus and deafness

Nat Genet. 1992 Aug;1(5):368-71. doi: 10.1038/ng0892-368.

Abstract

Non-insulin-dependent (type II) diabetes mellitus (NIDDM) is characterized by hyperglycaemia and insulin resistance, and affects nearly 5% of the general population. Inherited factors are important for its development, but the genes involved are unknown. We have identified a large pedigree in which NIDDM, in combination with a sensorineural hearing loss, is maternally inherited. The maternal inheritance and the observed decrease in mitochondrial enzyme activities of the respiratory chain indicate a genetic defect in the mitochondrial DNA. An A to G transition was identified at nucleotide 3,243, a conserved position in the mitochondrial gene for tRNA(Leu)(UUR). This mutation cosegregates with the disease in this family and is absent in controls, and indicates that a point mutation in mitochondrial DNA is a pathogenetic factor for NIDDM.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Cells, Cultured
  • Child
  • DNA, Mitochondrial / genetics*
  • DNA, Mitochondrial / isolation & purification
  • Deafness / complications
  • Deafness / genetics*
  • Diabetes Mellitus, Type 2 / complications
  • Diabetes Mellitus, Type 2 / genetics*
  • Female
  • Genetic Carrier Screening
  • HeLa Cells
  • Homozygote
  • Humans
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Muscles / metabolism
  • Oligodeoxyribonucleotides
  • Pedigree
  • Point Mutation*
  • Polymorphism, Restriction Fragment Length
  • RNA / genetics*
  • RNA, Mitochondrial
  • RNA, Transfer, Leu / genetics*
  • Restriction Mapping

Substances

  • DNA, Mitochondrial
  • Oligodeoxyribonucleotides
  • RNA, Mitochondrial
  • RNA, Transfer, Leu
  • RNA