Independence of growth factor induced plasminogen activator inhibitor type-1 (PAI-1) expression from the 4G/5G polymorphism of the PAI-1 gene

Thromb Haemost. 2003 Jul;90(1):36-42.

Abstract

Increased PAI-1 expression has been implicated in accelerating atherogenesis. Increases under some conditions are modulated by growth factors. Genetic factors such as the 4G/5G polymorphism in the promoter of the PAI-1 gene play a role under certain circumstances. The present study was designed to delineate for the first time interactions between growth factors and the 4G/5G polymorphism with respect to PAI-1 expression in human arterial smooth muscle cells (HASMC). HASMC were genotyped and exposed to growth factors. PAI-1 gene and protein expression were induced consistently by TGF-beta (up to 4.0-fold), PDGF (2.1-fold), TNF-alpha (1.7-fold), and thrombin (2.3-fold). Results were similar regardless of which genotype (4G/4G [n=9], 4G/5G [n=13], and 5G/5G [n=7]) was present. The induction of increased PAI-1 expression in human arterial smooth muscle cells by growth factors implicated in accelerated atherogenesis is independent of the PAI-1 4G/5G polymorphism. Accordingly, modulation of PAI-1 expression is likely to be influenced predominantly by environmental factors acting on, rather than genetic factors intrinsic to the PAI-1 promoter.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Arteriosclerosis / genetics
  • Arteriosclerosis / pathology
  • Cell Division / drug effects
  • Cells, Cultured / cytology
  • Cells, Cultured / drug effects
  • Gene Expression Regulation / drug effects*
  • Genotype
  • Growth Substances / pharmacology*
  • Growth Substances / physiology
  • Humans
  • Muscle, Smooth, Vascular / cytology
  • Muscle, Smooth, Vascular / drug effects*
  • Plasminogen Activator Inhibitor 1 / biosynthesis
  • Plasminogen Activator Inhibitor 1 / genetics*
  • Plasminogen Activator Inhibitor 1 / physiology
  • Platelet-Derived Growth Factor / pharmacology
  • Polymorphism, Genetic
  • Promoter Regions, Genetic / genetics
  • Thrombin / pharmacology
  • Tissue Plasminogen Activator / analysis
  • Transforming Growth Factor beta / pharmacology
  • Tumor Necrosis Factor-alpha / pharmacology
  • Urokinase-Type Plasminogen Activator / analysis

Substances

  • Growth Substances
  • Plasminogen Activator Inhibitor 1
  • Platelet-Derived Growth Factor
  • Transforming Growth Factor beta
  • Tumor Necrosis Factor-alpha
  • Thrombin
  • Tissue Plasminogen Activator
  • Urokinase-Type Plasminogen Activator