Nitric oxide alleviates oxidative damage in the green alga Chlorella pyrenoidosa caused by UV-B radiation

Folia Microbiol (Praha). 2003;48(3):389-93. doi: 10.1007/BF02931372.

Abstract

The effect of ultraviolet-B radiation (UV-B; 280-320 nm) on induction of nitric oxide was estimated in the suspensions of green alga Chlorella pyrenoidosa with or without the NO scavenger N-acetyl-L-cysteine, and reductants such as 1,4-dithiothreitol, glutathione (reduced form), and ascorbic acid. Exogenously added sodium nitroprusside (NO donor), glutathione, 1,4-dithiothreitol, and ascorbic acid were able to prevent chlorophyll loss mediated by UV-B. Addition of NO to algal suspensions irradiated by UV-B increased the activity of catalase and superoxide dismutase but lowered the activity of phenylalanine ammonia-lyase. UV-B thus appears to be a strong inducer of NO production, exogenously added NO and reductants protecting the green alga against UV-B-induced oxidative damage.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcysteine / pharmacology
  • Ascorbic Acid / pharmacology
  • Catalase / metabolism
  • Chlorella / enzymology
  • Chlorella / metabolism*
  • Chlorella / radiation effects*
  • Chlorophyll / metabolism
  • Dithiothreitol / pharmacology
  • Glutathione / pharmacology
  • Nitric Oxide / biosynthesis*
  • Nitroprusside / pharmacology
  • Oxidative Stress / drug effects
  • Oxidative Stress / physiology
  • Phenylalanine Ammonia-Lyase / metabolism
  • Superoxide Dismutase / metabolism
  • Ultraviolet Rays / adverse effects*

Substances

  • Chlorophyll
  • Nitroprusside
  • Nitric Oxide
  • Catalase
  • Superoxide Dismutase
  • Phenylalanine Ammonia-Lyase
  • Glutathione
  • Ascorbic Acid
  • Dithiothreitol
  • Acetylcysteine