[Transcriptional inactivation of RASSF1A in lung cancer and its clinical significance]

Zhonghua Yi Xue Za Zhi. 2003 Apr 10;83(7):569-71.
[Article in Chinese]

Abstract

Objective: To explore the relationship between the transcriptional expression of RASSF1A (Ras association domain family 1A gene) and oncogenesis and development of lung cancer.

Method: RT-PCR was used to detect the expression of RASSF1A mRNA in 47 human lung cancer tissues and matched 47 non-cancer tissues.

Results: (1). The RASSF1A mRNA was identified in all non-cancer tissues but not found in 53.2% carcinoma tissues. (2). The rate of loss of RASSF1A mRNA was significantly higher in the patients with positive lymph node metastasis (70.4%) than in those without lymph node metastasis (30.0%) (P < 0.01). (3). Much more frequent in advanced tumor tissues, loss of RASSF1A mRNA was correlated with TNM stage (P < 0.05). (4). No significant association of abnormal RASSF1A expression was identified with histological type, differentiation grade of tumors or age, sex, and smoking index of the patients (all P > 0.05).

Conclusion: RASSF1A is one of the new candidates of tumor suppressor genes. Loss or abnormal down-regulation of RASSF1A mRNA is a frequent event in lung carcinogenesis, which may play an important role in the malignant progression and prognosis of lung cancer.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • DNA Methylation
  • Genes, Tumor Suppressor
  • Humans
  • Lung Neoplasms / genetics*
  • Neoplasm Proteins / genetics*
  • RNA, Messenger / analysis
  • Transcription, Genetic*
  • Tumor Suppressor Proteins*

Substances

  • Neoplasm Proteins
  • RASSF1 protein, human
  • RNA, Messenger
  • Tumor Suppressor Proteins