Apolipoprotein A-I activates protein kinase C alpha signaling to phosphorylate and stabilize ATP binding cassette transporter A1 for the high density lipoprotein assembly

J Biol Chem. 2003 Nov 28;278(48):47890-7. doi: 10.1074/jbc.M306258200. Epub 2003 Sep 2.

Abstract

ATP-binding cassette transporter A1 (ABCA1) plays an essential role in the helical apolipoprotein-mediated assembly of high density lipoprotein, and the apolipoporteins stabilize ABCA1 against calpain-mediated degradation during the reaction ((2002) J. Biol. Chem. 277, 22426-22429). Protein kinase C (PKC) inhibitors suppressed both ABCA1 stabilization and cellular lipid release mediated by apolipoprotein A-I (apoA-I) but not ABCA1 increase by calpain inhibitors. The increase of ABCA1 and the cellular lipid release by apoA-I were both suppressed by a phosphatidylcholine phospholipase C (PC-PLC) inhibitor but not by the inhibitors of phosphatidylinositol-PLC and phosphatidylinositol 3-kinase. A protein phosphatase inhibitor further enhanced the ABCA1 increase by apoA-I. Biochemical and microscopic evidence indicated that apoA-I activated PKC alpha, and phosphorylation of ABCA1 was directly demonstrated by apoA-I via PKC. Finally, digestion of sphingomyelin increased ABCA1, and a PC-PLC inhibitor suppressed it. We conclude that apoA-I activates PKC alpha by PC-PLC-mediated generation of diacylglycerol initiated by the removal of cellular sphingomyelin ((2002) J. Biol. Chem. 277, 44709-44714), and subsequently phosphorylates and stabilizes ABCA1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ATP Binding Cassette Transporter 1
  • ATP-Binding Cassette Transporters / metabolism*
  • Apolipoprotein A-I / metabolism
  • Apolipoprotein A-I / physiology*
  • Apolipoproteins / metabolism
  • Blotting, Western
  • Calpain / antagonists & inhibitors
  • Cell Line
  • Diglycerides / metabolism
  • Dose-Response Relationship, Drug
  • Enzyme Activation
  • Enzyme Inhibitors / pharmacology
  • Fibroblasts / metabolism
  • Green Fluorescent Proteins
  • Humans
  • Lipid Metabolism
  • Lipoproteins, HDL / metabolism*
  • Luminescent Proteins / metabolism
  • Models, Biological
  • Phosphorylation
  • Protein Kinase C / antagonists & inhibitors
  • Protein Kinase C / metabolism*
  • Protein Kinase C-alpha
  • RNA, Messenger / metabolism
  • Recombinant Fusion Proteins / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Signal Transduction
  • Sphingomyelins / metabolism
  • Time Factors
  • Type C Phospholipases / antagonists & inhibitors

Substances

  • ABCA1 protein, human
  • ATP Binding Cassette Transporter 1
  • ATP-Binding Cassette Transporters
  • Apolipoprotein A-I
  • Apolipoproteins
  • Diglycerides
  • Enzyme Inhibitors
  • Lipoproteins, HDL
  • Luminescent Proteins
  • RNA, Messenger
  • Recombinant Fusion Proteins
  • Sphingomyelins
  • Green Fluorescent Proteins
  • PRKCA protein, human
  • Protein Kinase C
  • Protein Kinase C-alpha
  • Type C Phospholipases
  • phosphatidylcholine-specific phospholipase C
  • Calpain