BRAF and K-ras gene mutations in human pancreatic cancers

Cancer Lett. 2003 Sep 25;199(2):169-73. doi: 10.1016/s0304-3835(03)00384-7.

Abstract

We investigated the frequency of BRAF mutations in human pancreatic cancer specimens to determine its role in the development of pancreatic cancer. Nine pancreatic cancer samples without a K-ras codon 12 mutation and 19 with a K-ras mutation were included in the study. Analyses of the BRAF sequence revealed mutations in exon 15 (V599E) in two cases, both of which also exhibited a K-ras codon 12 mutation. No BRAF mutation was found in cases without a K-ras mutation. The BRAF V599E mutation was not found to be a major mutation in pancreatic cancers that had no K-ras codon 12 mutation.

MeSH terms

  • Adenocarcinoma / etiology
  • Adenocarcinoma / genetics
  • Adenocarcinoma, Mucinous / etiology
  • Adenocarcinoma, Mucinous / genetics
  • Aged
  • Aged, 80 and over
  • Carcinoma, Papillary / etiology
  • Carcinoma, Papillary / genetics
  • DNA Mutational Analysis
  • DNA Primers / chemistry
  • Female
  • Genes, ras / genetics*
  • Humans
  • Male
  • Middle Aged
  • Mutation / genetics*
  • Neoplasm Staging
  • Oncogene Proteins / genetics*
  • Pancreatic Neoplasms / etiology
  • Pancreatic Neoplasms / genetics*
  • Polymerase Chain Reaction
  • Proto-Oncogene Proteins B-raf
  • Survival Rate
  • Tumor Cells, Cultured

Substances

  • DNA Primers
  • Oncogene Proteins
  • BRAF protein, human
  • Proto-Oncogene Proteins B-raf