Enkephalin induces Ca2+ mobilization in single cells of bradykinin-sensitized differentiated neuroblastoma hybridoma (NG108-15) cells

Neurosci Lett. 1992 Dec 14;148(1-2):93-6. doi: 10.1016/0304-3940(92)90812-l.

Abstract

A study of the intracellular Ca2+ ([Ca2+]i) response of differentiated neuroblastoma x glioma hybrid cells (NG108-15 cell) to enkephalin (EK) was carried out by fura-2 video-imaging. EK alone did not influence [Ca2+]i in single cells. The opioid did, however, induce a marked [Ca2+]i rise, when the cells were incubated with bradykinin (BK) prior to the EK treatment. Such BK-assisted stimulation of the differentiated hybridoma cells by EK was completely abolished by pertussis toxin treatment. These results suggest that in single NG108-15 cells, EK induces Ca2+ mobilization which is assisted by cross-talk between the EK and BK receptor systems via a pertussis toxin-sensitive G protein.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bradykinin / pharmacology*
  • Bucladesine / pharmacology
  • Calcium / metabolism*
  • Calcium / pharmacology
  • Cell Differentiation / drug effects
  • Egtazic Acid / pharmacology
  • Enkephalins / pharmacology*
  • Fura-2
  • Glioma
  • Hybrid Cells
  • Kinetics
  • Mice
  • Microscopy, Fluorescence
  • Neuroblastoma
  • Pertussis Toxin
  • Rats
  • Time Factors
  • Video Recording
  • Virulence Factors, Bordetella / pharmacology

Substances

  • Enkephalins
  • Virulence Factors, Bordetella
  • Egtazic Acid
  • Bucladesine
  • Pertussis Toxin
  • Bradykinin
  • Calcium
  • Fura-2