Lifelong ethanol consumption enhances the age-related changes in rat sympathetic neurons

Mech Ageing Dev. 1992 Apr;63(2):193-205. doi: 10.1016/0047-6374(92)90065-l.

Abstract

The effects of aging and chronic ethanol administration on the histochemical and morphometric features of rat superior cervical ganglion were studied in a rat strain selected for voluntary alcohol consumption. Ethanol was administered to the experimental group ad libitum (10% v/v in drinking water) from 3 months to 28 months of age, the average ethanol intake being 6.4-5.4 g/kg per day. The sympathetic neurons of the ethanol consuming rats showed several signs of enhanced degeneration, e.g. decreased neuronal packing density, increased amount of age-pigment and decreased intensity of catecholamine histofluorescence and tyrosine hydroxylase immunoreactivity. The results may indicate a selective vulnerability of peripheral sympathetic neurons rather than a universal accelerated aging due to chronic ethanol exposure.

MeSH terms

  • Aging / metabolism
  • Aging / pathology*
  • Alcoholism / metabolism
  • Alcoholism / pathology*
  • Animals
  • Catecholamines / metabolism
  • Male
  • Nerve Degeneration
  • Neurons / metabolism
  • Neurons / pathology
  • Pigments, Biological / metabolism
  • Rats
  • Sympathetic Nervous System / metabolism
  • Sympathetic Nervous System / pathology*
  • Tyrosine 3-Monooxygenase / metabolism

Substances

  • Catecholamines
  • Pigments, Biological
  • Tyrosine 3-Monooxygenase