Development of CD30+ lymphoproliferative disease in mice lacking interferon regulatory factor-1

Oncogene. 2003 Sep 18;22(40):6166-76. doi: 10.1038/sj.onc.1206563.

Abstract

Human lymphomas continue to represent a major challenge in oncology, and in particular occur at very high frequencies in AIDS patients. We report here the development of a CD30+ lymphoproliferative disease in mice lacking the proapoptotic transcription factor, interferon regulatory factor-1. These mice most closely represent a model of human anaplastic large-cell lymphoma (ALCL). This mouse model of lymphoma will likely be useful in understanding the development of ALCL and in understanding the development of other closely related CD30+ forms of lymphoma, such as CD30+ Hodgkin's disease and CD30+ cutaneous T-cell lymphoma. This mouse model will also be useful in testing therapies for different forms of CD30+ lymphoma, in particular anti-CD30-based therapies.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • DNA-Binding Proteins / genetics*
  • Immunohistochemistry
  • Interferon Regulatory Factor-1
  • Ki-1 Antigen / metabolism*
  • Lymph Nodes / metabolism
  • Lymph Nodes / pathology
  • Lymphatic Diseases / metabolism
  • Lymphatic Diseases / pathology
  • Lymphoma, Large-Cell, Anaplastic / metabolism*
  • Lymphoma, Large-Cell, Anaplastic / pathology
  • Lymphoma, Large-Cell, Anaplastic / ultrastructure
  • Lymphoproliferative Disorders / genetics*
  • Lymphoproliferative Disorders / metabolism*
  • Lymphoproliferative Disorders / pathology
  • Mice
  • Mice, Knockout
  • Phosphoproteins / genetics*
  • Retinoblastoma Protein / genetics
  • Time Factors
  • Transcription Factors / genetics

Substances

  • DNA-Binding Proteins
  • Interferon Regulatory Factor-1
  • Irf1 protein, mouse
  • Ki-1 Antigen
  • Phosphoproteins
  • Retinoblastoma Protein
  • Transcription Factors