[3H]zonisamide binding in rat brain

Med J Osaka Univ. 1990 Mar;39(1-4):19-22.

Abstract

We previously reported that zonisamide inhibits both [3H]flunitrazepam and [3H]muscimol binding in rat brain. In the present study, [3H]zonisamide was found to bind in a saturable fashion to the crude synaptosomal fraction of whole rat brain. Linear regression analysis of the binding data in the Scatchard plot indicated a Kd of 90 nM, and a maximal binding capacity of 1.40 x 10(3) fmol/mg protein. Displacement studies revealed an inhibitory effect of clonazepam and an enhancement effect of GABA on specific [3H]zonisamide binding. These results suggest that specific [3H]zonisamide binding sites may have a tight correlationship with benzodiazepine receptors in rat brain.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Anticonvulsants / metabolism*
  • Brain / metabolism*
  • Clonazepam / pharmacology
  • Dose-Response Relationship, Drug
  • Isoxazoles / metabolism*
  • Male
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, GABA-A / metabolism*
  • Synaptosomes / metabolism*
  • Zonisamide
  • gamma-Aminobutyric Acid / pharmacology

Substances

  • Anticonvulsants
  • Isoxazoles
  • Receptors, GABA-A
  • Zonisamide
  • gamma-Aminobutyric Acid
  • Clonazepam