Interleukin-10 (IL-10) inhibits the induction of nitric oxide synthase by interferon-gamma in murine macrophages

Biochem Biophys Res Commun. 1992 Feb 14;182(3):1155-9. doi: 10.1016/0006-291x(92)91852-h.

Abstract

A murine macrophage cell line, J774, expresses high levels of the enzyme nitric oxide synthase (NOS) and produces large amounts of nitric oxide (NO) when activated with recombinant interferon (IFN)-gamma and a low concentration of LPS (10 ng/ml). Both the expression of NOS and the production of NO were inhibited by recombinant IL-10 in a dose-dependent manner. The inhibition was effective only when the cells were pretreated with IL-10; addition of IL-10 at the same time or after IFN-gamma activation was without effect. These results demonstrate that IL-10, a product of Th2 (helper T lymphocyte 2) cells, can antagonise the function of IFN-gamma, a product of Th1 cells, by modulating the mechanism of synthesis of nitric oxide in the macrophages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Oxidoreductases / biosynthesis*
  • Animals
  • Cell Line
  • Enzyme Induction / drug effects
  • Interferon-gamma / antagonists & inhibitors
  • Interferon-gamma / pharmacology*
  • Interleukin-10 / pharmacology*
  • Kinetics
  • Macrophages / drug effects
  • Macrophages / enzymology*
  • Mice
  • Nitric Oxide / metabolism
  • Nitric Oxide Synthase
  • Recombinant Proteins / pharmacology

Substances

  • Recombinant Proteins
  • Interleukin-10
  • Nitric Oxide
  • Interferon-gamma
  • Nitric Oxide Synthase
  • Amino Acid Oxidoreductases