Myasthenogenicity of a human acetylcholine receptor alpha-subunit peptide: morphology and immunology

Muscle Nerve. 1992 Mar;15(3):282-7. doi: 10.1002/mus.880150305.

Abstract

Each of 10 rats inoculated with a synthetic peptide comprising residues 125-147 (without a disulfide bond) of human acetylcholine receptor (AChR) alpha-subunit (H alpha) had deposits of IgG and C3 (immune complexes) and showed morphological changes in the fine structure at the motor end-plates 5 weeks after a single immunization. Antibody to the H alpha peptides was elevated 1 week after immunization, but, antibody levels to solubilized human or rat AChR were very low in 8 of the 10 rats. These results suggest that the immune response to peptide H alpha is the myasthenogenic site, which induces morphological change at the end-plates.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Epitopes / immunology
  • Female
  • Immunization
  • Microscopy, Electron
  • Motor Endplate / immunology
  • Motor Endplate / ultrastructure*
  • Myasthenia Gravis / immunology*
  • Myasthenia Gravis / physiopathology
  • Peptide Fragments* / immunology
  • Rats
  • Rats, Inbred Lew
  • Receptors, Cholinergic* / immunology
  • Receptors, Nicotinic*

Substances

  • Epitopes
  • Peptide Fragments
  • Receptors, Cholinergic
  • Receptors, Nicotinic
  • acetylcholine receptor alpha-subunit (125-148)