c-myc expression is down-regulated by cell-cell and cell-extracellular matrix contacts in normal hepatocytes, but not in hepatoma cells

Biochem Biophys Res Commun. 1992 Apr 30;184(2):825-31. doi: 10.1016/0006-291x(92)90664-7.

Abstract

Primary culture of adult rat hepatocytes resulted in marked increase of c-myc expression within a few hours. The high level of c-myc mRNA was maintained throughout culture on collagen-coated dishes, but decreased greatly with time during culture on collagen-gel or matrigel. Expression of c-myc was also down-regulated at high cell density. The decrease in its expression appeared closely related to inhibitions of DNA synthesis and cell spreading. In contrast, hepatoma H4TG cells showed a high level of c-myc expression which was not affected by culture on any extracellular matrices examined or by the cell density. These results suggest that up-regulation of expression of the c-myc gene is linked to G0 to G1 transition during cell cycle progression, which in normal hepatocytes is strictly regulated by cell-cell and cell-extracellular matrix interactions, but that this control mechanism is defective in malignant hepatic tumor cells.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Cell Communication*
  • Cell Line
  • Cells, Cultured
  • DNA Replication*
  • Extracellular Matrix / physiology*
  • Gene Expression Regulation*
  • Gene Expression Regulation, Neoplastic*
  • Genes, myc*
  • Kinetics
  • Liver / physiology*
  • Liver Neoplasms, Experimental / genetics*
  • Male
  • RNA / genetics
  • RNA / isolation & purification
  • Rats
  • Rats, Inbred Strains

Substances

  • RNA