Insulin-like growth factor-I and follicle-stimulating hormone suppress insulin-like growth factor binding protein-1 secretion by human granulosa-luteal cells

J Clin Endocrinol Metab. 1992 Sep;75(3):969-71. doi: 10.1210/jcem.75.3.1381377.

Abstract

Local regulation of insulin-like growth factor binding protein-1 (IGFBP-1) production in the human ovarian follicle was investigated using cultured human granulosa-luteal cells. Both insulin-like growth factor-I (IGF-I) and follicle-stimulating hormone (FSH) exerted a dual effect on granulosa cells: while estradiol (E2) production was increased by both stimulants, the addition of either of the two hormones led to a reduction in IGFBP-1 secretion by more than 50%. Inhibition of IGFBP-1 production in response to IGF-I was dose-dependent,with the highest effect observed at 5 nM IGF-I. A significant correlation was found between the increase in E2 and inhibition of IGFBP-1 secretion in response to IGF-I. These observations may suggest a novel mechanism, at the follicular level, by which FSH and IGF-I amplify the IGF-I effect in the ovarian follicular cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Carrier Proteins / antagonists & inhibitors*
  • Carrier Proteins / metabolism
  • Cells, Cultured
  • Corpus Luteum / cytology
  • Corpus Luteum / metabolism*
  • Dose-Response Relationship, Drug
  • Estradiol / metabolism
  • Female
  • Follicle Stimulating Hormone / pharmacology*
  • Granulosa Cells / metabolism*
  • Humans
  • Insulin-Like Growth Factor Binding Protein 1
  • Insulin-Like Growth Factor I / pharmacology*

Substances

  • Carrier Proteins
  • Insulin-Like Growth Factor Binding Protein 1
  • Estradiol
  • Insulin-Like Growth Factor I
  • Follicle Stimulating Hormone