Structural basis of the intrasteric regulation of myosin light chain kinases

Science. 1992 Oct 2;258(5079):130-5. doi: 10.1126/science.1439761.

Abstract

The smooth muscle myosin light chain kinase (smMLCK) catalytic core was modeled by using the crystallographic coordinates of the cyclic AMP-dependent protein kinase catalytic subunit (cAPK) and a bound pseudosubstrate inhibitor peptide, PKI(5-24). Despite only 30% identity in amino acid sequence, the MLCK sequence can be readily accommodated in this structure. With the exception of the short B-helix, all major elements of secondary structure in the core are very likely conserved. The active site of the modeled MLCK complements the known requirements for peptide substrate recognition. MLCK contains a pseudosubstrate sequence that overlaps the calmodulin binding domain and has been proposed to act as an intrasteric inhibitor and occupy the substrate binding site in the absence of Ca(2+)-calmodulin. The pseudosubstrate sequence can be modeled easily into the entire backbone of PKI(5-24). The results demonstrate that the intrasteric model for regulation of MLCK by intramolecular competitive inhibition is structurally plausible.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Binding Sites
  • Chromosome Mapping
  • Crystallography
  • Gene Expression Regulation, Enzymologic*
  • Models, Molecular
  • Molecular Sequence Data
  • Molecular Structure
  • Myosin-Light-Chain Kinase / chemistry*
  • Oligopeptides / genetics
  • Oligopeptides / metabolism
  • Peptide Fragments
  • Peptides / genetics
  • Peptides / metabolism
  • Protein Binding / physiology
  • Protein Kinases / chemistry
  • Sequence Alignment
  • Sequence Homology

Substances

  • Oligopeptides
  • Peptide Fragments
  • Peptides
  • protein kinase inhibitor peptide
  • kemptide
  • Protein Kinases
  • Myosin-Light-Chain Kinase