Bidirectional ventricular tachycardia and channelopathy

Am J Cardiol. 2003 Oct 15;92(8):991-5. doi: 10.1016/s0002-9149(03)00987-1.

Abstract

Based on similarity of electrocardiographic features, bidirectional ventricular tachycardia has been considered a variant of long QT syndrome. Genes causing long QT syndrome were used as candidate genes in 4 patients with bidirectional ventricular tachycardia. In 2 patients, we identified a common low penetrance HERG allele (R1047L) with an intermediate biophysical phenotype.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Alleles
  • Cation Transport Proteins*
  • Child, Preschool
  • DNA-Binding Proteins*
  • ERG1 Potassium Channel
  • Electrophysiologic Techniques, Cardiac
  • Ether-A-Go-Go Potassium Channels
  • Female
  • Humans
  • Long QT Syndrome / genetics*
  • Male
  • Phenotype
  • Potassium Channels / genetics*
  • Potassium Channels, Voltage-Gated*
  • Sodium Channels / genetics
  • Tachycardia, Ventricular / genetics*
  • Trans-Activators*
  • Transcriptional Regulator ERG

Substances

  • Cation Transport Proteins
  • DNA-Binding Proteins
  • ERG protein, human
  • ERG1 Potassium Channel
  • Ether-A-Go-Go Potassium Channels
  • KCNH2 protein, human
  • KCNH6 protein, human
  • Potassium Channels
  • Potassium Channels, Voltage-Gated
  • Sodium Channels
  • Trans-Activators
  • Transcriptional Regulator ERG