Impact of endoscopically minimal involvement on IL-8 mRNA expression in esophageal mucosa of patients with non-erosive reflux disease

World J Gastroenterol. 2003 Dec;9(12):2801-4. doi: 10.3748/wjg.v9.i12.2801.

Abstract

Aim: Little has been known about the pathogenesis of non-erosive reflux disease (NERD). Recent studies have implicated interleukin 8 (IL-8) in the development and progression of gastroesophogeal reflux disease (GERD). The purpose of this study was to determine IL-8 RNA expression levels in NERD patients with or without subtle mucosal changes.

Methods: We studied 26 patients with NERD and 13 asymptomatic controls. Biopsy sample was taken from the esophagus 3 cm above the gastroesophageal junction and snap frozen for measurement of IL-8 mRNA levels by real-time quantitative polymerase chain reaction (PCR). We also examined mRNA expression of IL-8 receptors, CXCR-1 and -2 by reverse transcriptase PCR. The patients were endoscopically classified into grade M (mucosal color changes without visible mucosal break) and N (neither minimal involvement nor mucosal break) of the modified Los Angeles classification.

Results: The relative IL-8 mRNA expression levels were significantly higher in esophageal mucosa of NERD patients than those in esophageal mucosa of the controls. There was a significant difference in IL-8 mRNA levels between grades M and N. The CXCR-1 and -2 mRNAs were constitutively expressed in esophageal mucosa.

Conclusion: Our results suggest that high IL-8 levels in esophageal mucosa may be involved in the pathogenesis of NERD through interaction with its receptors. NERD seems to be composed of a heterogeneous population in terms of not only endoscopically minimal involvement but also immune and inflammatory processes.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Aged
  • Alcohol Drinking
  • Base Sequence
  • DNA Primers
  • Endoscopy, Digestive System
  • Female
  • Gastroesophageal Reflux / classification
  • Gastroesophageal Reflux / diagnosis
  • Gastroesophageal Reflux / genetics*
  • Gastroesophageal Reflux / immunology*
  • Gene Expression Regulation / immunology
  • Helicobacter Infections / epidemiology
  • Helicobacter pylori
  • Hernia, Hiatal / epidemiology
  • Humans
  • Interleukin-8 / genetics*
  • Male
  • Middle Aged
  • RNA, Messenger / genetics*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Risk Factors
  • Smoking

Substances

  • DNA Primers
  • Interleukin-8
  • RNA, Messenger