Abstract
4-1BB (CD137) is a costimulatory molecule involved in the activation and survival of CD4, CD8, and natural killer cells. Although a great deal has been learned as to how 4-1BB-mediated signaling governs the immunity of conventional T cells, the functional role of 4-1BB in the context of CD4(+)CD25(+) regulatory T cell (Tr) activation is largely unknown. Using 4-1BB-intact and -deficient mice, we investigated the effect of the 4-1BB/4-1BB ligand pathway on the suppressive function of Tr cells. Our data indicate that although 4-1BB is expressed on Tr cells, its contribution to their proliferation is minimal. We also showed that signaling through the 4-1BB receptor inhibited the suppressive function of Tr cells in vitro and in vivo. It is interesting that anti-4-1BB-mediated but not anti-GITR-directed inhibition was more potent when Tr cells were preactivated. Collectively, these data indicate that 4-1BB signaling is critical in Tr cell immunity.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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4-1BB Ligand
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Animals
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Antigens, CD
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CD4-Positive T-Lymphocytes / immunology
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CD4-Positive T-Lymphocytes / transplantation
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Female
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Graft vs Host Disease / etiology
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Immunity*
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Lymphocyte Activation / immunology*
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Lymphocyte Transfusion / adverse effects
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Mice
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Mice, Knockout
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Receptors, Interleukin-2
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Receptors, Nerve Growth Factor / deficiency
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Receptors, Nerve Growth Factor / immunology
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Receptors, Nerve Growth Factor / physiology*
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Receptors, Tumor Necrosis Factor / deficiency
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Receptors, Tumor Necrosis Factor / immunology
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Receptors, Tumor Necrosis Factor / physiology*
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Signal Transduction / immunology
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T-Lymphocytes, Helper-Inducer / immunology*
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T-Lymphocytes, Helper-Inducer / transplantation
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Tumor Necrosis Factor Receptor Superfamily, Member 9
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Tumor Necrosis Factor-alpha / metabolism
Substances
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4-1BB Ligand
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Antigens, CD
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Receptors, Interleukin-2
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Receptors, Nerve Growth Factor
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Receptors, Tumor Necrosis Factor
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Tnfrsf9 protein, mouse
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Tnfsf9 protein, mouse
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Tumor Necrosis Factor Receptor Superfamily, Member 9
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Tumor Necrosis Factor-alpha