Involvement of tumor necrosis factor-alpha and interleukin-8 in antigen-induced arthritis of the rabbit temporomandibular joint

J Oral Pathol Med. 2004 Feb;33(2):102-10. doi: 10.1111/j.1600-0714.2004.00008.x.

Abstract

Background: In temporomandibular joint (TMJ) arthritis, knowledge is limited about the source of the inflammatory mediators. The aim of this study was to investigate the immunohistochemical role of tumor necrosis factor-alpha (TNF-alpha) and interleukin-8 (IL-8) in the development of the antigen-induced arthritis of the rabbit TMJ.

Methods: Unilateral TMJ arthritis was induced in 28 adult rabbits. From 6 h to 6 weeks after induction of arthritis, the topology of TNF-alpha and IL-8 was observed.

Results: Positive reaction for TNF-alpha of synovial cells was observed within 3 days after induction and at 3 weeks after induction. TNF-alpha positive vascular endothelial cells and chondrocytes were identified throughout the observation period. IL-8 was detected only during the acute stage.

Conclusions: The cytokines TNF-alpha and IL-8 were observed in specific cells depending on the stage. TNF-alpha was particularly related with angiogenesis and cartilage destruction and IL-8 was involved in the acute stage of inflammation.

MeSH terms

  • Animals
  • Arthritis, Experimental / metabolism*
  • Arthritis, Experimental / pathology
  • Cartilage, Articular / metabolism
  • Cartilage, Articular / pathology
  • Chondrocytes / metabolism
  • Immunohistochemistry
  • Interleukin-8 / metabolism*
  • Male
  • Neovascularization, Pathologic
  • Rabbits
  • Temporomandibular Joint Disorders / immunology
  • Temporomandibular Joint Disorders / metabolism*
  • Temporomandibular Joint Disorders / pathology
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Interleukin-8
  • Tumor Necrosis Factor-alpha