An autosome-wide search using longitudinal data for loci linked to type 2 diabetes progression

BMC Genet. 2003 Dec 31;4 Suppl 1(Suppl 1):S8. doi: 10.1186/1471-2156-4-S1-S8.

Abstract

A genome-wide screen was conducted for type 2 diabetes progression genes using measures of elevated fasting glucose levels as quantitative traits from the offspring enrolled in the Framingham Heart Study. We analyzed young (20-34 years) and old (>or= 35 years) subjects separately, using single-point and multipoint sibpair analysis, because of the possible differential impact of progression on the groups of interest. We observed significant linkage with change in fasting glucose levels on 1q25-32 (p = 5.21 x 10(-8)), 3p26.3-21.31 (p = 1 x 10(-11)), 8q23.1-24.13 (p = 2.94 x 10(-6)), 9p24.1-21.3 (p = 7 x 10(-7)), and 18p11.31-q22.1 (p < 10(-11)). The evidence for linkage on chromosomes 8 and 18 was consistent for the subset of study participants aged 43 through 55 years.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Adult Children
  • Blood Glucose / genetics
  • Chromosomes, Human, Pair 1 / genetics
  • Chromosomes, Human, Pair 18 / genetics
  • Chromosomes, Human, Pair 3 / genetics
  • Chromosomes, Human, Pair 8 / genetics
  • Chromosomes, Human, Pair 9 / genetics
  • Cohort Studies
  • Data Interpretation, Statistical
  • Diabetes Mellitus, Type 2 / blood
  • Diabetes Mellitus, Type 2 / genetics*
  • Diabetes Mellitus, Type 2 / pathology*
  • Fasting / blood
  • Female
  • Genetic Linkage / genetics*
  • Genome, Human
  • Humans
  • Longitudinal Studies
  • Male
  • Middle Aged
  • Quantitative Trait Loci / genetics*
  • Siblings

Substances

  • Blood Glucose