A novel non-neuronal hSK3 isoform with a dominant-negative effect on hSK3 currents

Cell Physiol Biochem. 2004;14(1-2):23-30. doi: 10.1159/000076923.

Abstract

We have identified a hSK3-transcript, hSK3_ex1c, which is generated by alternative splicing. Isoform hSK3_ex1c lacks the cytosolic N-terminus and the first transmembrane helix and is exclusively expressed in non-neuronal tissues. hSK3 transfected tsA cells showed a Ca2+-activated K+ current in patch-clamp experiments, whereas hSK3_ex1c transfected cells and cells co-transfected with both isoforms did not. We fused both isoforms to fluorescence proteins and observed hSK3 localization predominantly in the plasma membrane. The co-expression of hSK3 + hSK3_ex1c resulted in their cytoplasmic co-localization. Thus, hSK3_ex1c has a dominant-negative effect on hSK3 by preventing its transport into the plasma membrane.

MeSH terms

  • Alternative Splicing
  • Amino Acid Sequence
  • Cell Line
  • Cell Membrane / physiology
  • Exons
  • Gene Expression
  • Genes, Dominant / genetics*
  • Humans
  • Molecular Sequence Data
  • Potassium Channels / genetics
  • Potassium Channels / physiology*
  • Potassium Channels, Calcium-Activated / genetics
  • Potassium Channels, Calcium-Activated / physiology
  • Protein Isoforms / genetics
  • Protein Isoforms / physiology*
  • Small-Conductance Calcium-Activated Potassium Channels

Substances

  • Potassium Channels
  • Potassium Channels, Calcium-Activated
  • Protein Isoforms
  • Small-Conductance Calcium-Activated Potassium Channels

Associated data

  • GENBANK/AA255937
  • GENBANK/AF336797
  • GENBANK/AJ251016