Selective CD27+ (memory) B cell reduction and characteristic B cell alteration in drug-naive and HAART-treated HIV type 1-infected patients

AIDS Res Hum Retroviruses. 2004 Feb;20(2):219-26. doi: 10.1089/088922204773004941.

Abstract

To investigate HIV-1-related B cell disorders, the quantity of CD27 positive (CD27+) B cells and their CD38, CD95, and bcl-2 intensities were examined by flow cytometry analysis in 16 drug-naive patients, 27 highly active antiretroviral therapy (HAART)-treated patients, and 20 uninfected controls. CD27+ B cells have been recognized as memory B cells. The mean percentage of CD27+ B cells was significantly lower in drug-naive patients (11.9%) and in HAART-treated patients (16.1%) than in controls (31.4%) (p < 0.01). The intensities of CD38 and CD95 on CD27+ B cells were higher in drug-naive patients than in controls (p < 0.01 in CD95). The intensity of CD95 on CD27+ B cells in HAART-treated patients was lower than that of drug-naive patients, but significantly higher than that of controls (p < 0.01). The intensity of bcl-2 on CD27+ B cells was equivalent among the three groups. These findings suggest that disturbance of peripheral B cell composition, exemplified by CD27+ B cell reduction, exists in both drug-naive and HAART-treated HIV-1-infected patients. In addition, the augmented apoptotic state of CD27+ B cells found in HAART-treated patients with undetectable viral loads, indicated by CD95 elevation, suggests that some HIV-1-related B cell disorders last for years after effective antiviral therapy.

MeSH terms

  • ADP-ribosyl Cyclase / metabolism
  • ADP-ribosyl Cyclase 1
  • Adult
  • Antigens, CD / metabolism
  • Antiretroviral Therapy, Highly Active*
  • B-Lymphocyte Subsets / immunology*
  • Case-Control Studies
  • HIV Infections / drug therapy*
  • HIV Infections / immunology*
  • HIV-1
  • Humans
  • Immunologic Memory
  • Membrane Glycoproteins
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Tumor Necrosis Factor Receptor Superfamily, Member 7 / metabolism*
  • fas Receptor / metabolism

Substances

  • Antigens, CD
  • Membrane Glycoproteins
  • Proto-Oncogene Proteins c-bcl-2
  • Tumor Necrosis Factor Receptor Superfamily, Member 7
  • fas Receptor
  • ADP-ribosyl Cyclase
  • CD38 protein, human
  • ADP-ribosyl Cyclase 1