Susceptibility to transglutaminase of gliadin peptides predicted by a mass spectrometry-based assay

FEBS Lett. 2004 Mar 26;562(1-3):177-82. doi: 10.1016/S0014-5793(04)00231-5.

Abstract

A peptidomics approach was developed to identify transglutaminase-susceptible Q residues within a pepsin-trypsin gliadin digest. Based on tagging with a monodansylcadaverine fluorescent probe, six alpha/beta-, gamma-gliadin, and low molecular weight glutenin peptides were identified by nanospray tandem mass spectrometry. In functioning as an acyl acceptor, tissue transglutaminase was able to form complexes with the glutamine-rich gliadin peptides, whereas by lowering pH, the peptides were deamidated by transglutaminase at the same Q residues, which were previously transamidated. The main common feature shared by the peptides was the consensus sequence Q-X-P. Our findings offer relevant information for the understanding of how dietary peptides interact with the host organism in celiac disease.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Autoantibodies / immunology
  • Gliadin / chemistry
  • Gliadin / genetics
  • Gliadin / immunology
  • Gliadin / metabolism*
  • Guinea Pigs
  • Humans
  • Mass Spectrometry*
  • Molecular Sequence Data
  • Peptides / chemistry
  • Peptides / genetics
  • Peptides / immunology
  • Peptides / metabolism*
  • Transglutaminases / metabolism*

Substances

  • Autoantibodies
  • Peptides
  • Gliadin
  • Transglutaminases