Human brain contains a metalloprotease that converts big endothelin-1 to endothelin-1 and is inhibited by phosphoramidon and EDTA

Br J Pharmacol. 1992 Jul;106(3):505-6. doi: 10.1111/j.1476-5381.1992.tb14364.x.

Abstract

Incubation of big endothelin-1 (bET-1) with protein derived from the detergent-extracted 100,000 g pellet prepared from human brain tissue resulted in the formation of endothelin-1 (ET-1) at a rate of 90 fmol mg-1 protein min-1. This formation was inhibited in a concentration-dependent manner by either phosphoramidon or EDTA, with half-maximal inhibitory concentrations of 2 and 20 microM, respectively. No conversion of big endothelin-3 (bET-3) to endothelin-3 (ET-3) was detected under the same conditions. These results show the presence in the human brain of a metalloprotease-like enzymatic activity which selectively converts bET-1 and ET-1. Together with earlier reports of mRNA for ET-1 this suggests the presence of the entire synthetic pathway for ET-1 in human brain.

MeSH terms

  • Brain / enzymology*
  • Dose-Response Relationship, Drug
  • Edetic Acid / pharmacology*
  • Endothelin-1
  • Endothelins / metabolism*
  • Glycopeptides / pharmacology*
  • Humans
  • Metalloendopeptidases / analysis*
  • Metalloendopeptidases / antagonists & inhibitors
  • Neprilysin / antagonists & inhibitors
  • Protein Precursors / metabolism*

Substances

  • Endothelin-1
  • Endothelins
  • Glycopeptides
  • Protein Precursors
  • Edetic Acid
  • Metalloendopeptidases
  • Neprilysin
  • phosphoramidon