Alzheimer's disease, genes, and environment: the value of international studies

Can J Psychiatry. 2004 Feb;49(2):92-9. doi: 10.1177/070674370404900203.

Abstract

Objective: To describe the construction of a disease model incorporating both genetic an environmental factors in the etiology of Alzheimer's disease (AD), using data generated from the Indianapolis-Ibadan dementia project (I-IDP).

Method: The I-IDP is a longitudinal comparative study of the prevalence and incidence o dementia in 2 communities: elderly African Americans living in Indianapolis, Indiana, an Yoruba living in Ibadan, Nigeria.

Results: African Americans are more than twice as likely as Yoruba to develop AD. Possible explanations for this finding include genetic factors: the possession of the apolipoprotein E epsilon4 allele does not increase risk for AD among Yoruba but confers a sligh increase in AD risk for African Americans. As well, environmental factors may play a role: African Americans have a higher risk of vascular risk factors than do Yoruba.

Conclusions: International comparative studies, particularly those involving population from developing and developed countries, offer a unique opportunity for applying new in formation regarding population genetics to traditional AD risk factor research.

Publication types

  • Comparative Study
  • Multicenter Study
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Aged
  • Alleles
  • Alzheimer Disease / diagnosis
  • Alzheimer Disease / epidemiology
  • Alzheimer Disease / genetics*
  • Apolipoprotein E4
  • Apolipoproteins E / genetics
  • Black People / genetics*
  • Black People / statistics & numerical data
  • Black or African American
  • Cross-Cultural Comparison*
  • Cross-Sectional Studies
  • Dementia, Vascular / diagnosis
  • Dementia, Vascular / epidemiology
  • Dementia, Vascular / genetics*
  • Genetic Variation / genetics
  • Genetics, Population
  • Humans
  • Incidence
  • Indiana
  • Life Style
  • Longitudinal Studies
  • Nigeria
  • Phenotype
  • Risk Factors
  • Social Environment*
  • Urban Population / statistics & numerical data*

Substances

  • Apolipoprotein E4
  • Apolipoproteins E