Estrogen negatively regulates rat gonadotropin releasing hormone (rGnRH) promoter activity in transfected placental cells

Mol Cell Endocrinol. 1992 Jul;86(1-2):1-10. doi: 10.1016/0303-7207(92)90169-7.

Abstract

To dissect the functional architecture of the rat gonadotropin releasing hormone (GnRH) gene promoter and its regulation by estrogen, gene transfer studies were performed in a placental cell line. 5'-Deletional constructs demonstrated that cis-acting elements important for rGnRH promoter activity in placenta were contained within a region of the proximal promoter between -73 and -16 bp upstream of the transcription initiation site. In addition, an inhibitory region was found from -903 to -424. Deletion of sequences to -424 which removed the inhibitory region, produced a promoter fragment which exhibited a 50% inhibition of GnRH promoter activity in the presence of estrogen (E) and cotransfected estrogen receptor. Negative regulation by E was retained in constructs deleted to -73 bp. We conclude that sequences important for placental cell expression and estrogen regulation of rGnRH lie within the region from -73 to -16. In addition, potential repressor sequences active in placental cells are present between -903 to 424.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Base Sequence
  • Cell Line
  • Choriocarcinoma / pathology
  • Depression, Chemical
  • Estrogens / pharmacology*
  • Female
  • Gene Expression Regulation / drug effects*
  • Gonadotropin-Releasing Hormone / biosynthesis
  • Gonadotropin-Releasing Hormone / genetics*
  • Humans
  • Hypothalamus / cytology
  • Hypothalamus / metabolism
  • Mice
  • Molecular Sequence Data
  • Neurons / metabolism
  • Placenta / metabolism*
  • Placenta / pathology
  • Promoter Regions, Genetic / drug effects*
  • RNA, Messenger / analysis
  • Rats
  • Receptors, Estrogen / genetics
  • Receptors, Estrogen / metabolism
  • Recombinant Fusion Proteins / biosynthesis
  • Regulatory Sequences, Nucleic Acid*
  • Transcription, Genetic / drug effects
  • Transfection
  • Tumor Cells, Cultured / drug effects
  • Tumor Cells, Cultured / metabolism
  • Uterine Neoplasms / pathology

Substances

  • Estrogens
  • RNA, Messenger
  • Receptors, Estrogen
  • Recombinant Fusion Proteins
  • Gonadotropin-Releasing Hormone