Abstract
The gene encoding dysbindin-1 has recently been implicated in susceptibility to schizophrenia. In this issue of the JCI, Talbot et al. show that, contrary to expectations, dysbindin-1 is located presynaptically in glutamatergic neurons and is reduced at these locations in schizophrenia. Further studies of dysbindin-1 and the proteins with which it interacts can be expected to throw light on the pathogenesis of schizophrenia.
MeSH terms
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Brain / metabolism
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Carrier Proteins / genetics*
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Carrier Proteins / metabolism
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Dysbindin
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Dystrophin-Associated Proteins
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Genetic Predisposition to Disease
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Glutamic Acid / metabolism
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Hippocampus / cytology
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Hippocampus / metabolism
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Humans
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Membrane Transport Proteins*
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Models, Biological
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Neurons / metabolism*
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Neurons / pathology
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Presynaptic Terminals / metabolism
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Schizophrenia / etiology
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Schizophrenia / genetics
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Schizophrenia / pathology*
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Tissue Distribution
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Vesicular Glutamate Transport Protein 1
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Vesicular Transport Proteins*
Substances
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Carrier Proteins
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DTNBP1 protein, human
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Dysbindin
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Dystrophin-Associated Proteins
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Membrane Transport Proteins
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SLC17A7 protein, human
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Vesicular Glutamate Transport Protein 1
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Vesicular Transport Proteins
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Glutamic Acid