Inhalation toxicology and carcinogenesis studies of propylene glycol mono-t-butyl ether in rats and mice

Toxicology. 2004 Jun 1;199(1):1-22. doi: 10.1016/j.tox.2003.12.020.

Abstract

Propylene glycol mono-t-butyl ether (PGMBE) is used as a solvent in a variety of commercial applications. Male and female F344/N rats and B6C3F(1) mice were exposed to PGMBE by whole-body inhalation for 2 or 14 weeks (0, 75, 150, 300, 600, or 1200 ppm) or 2 years (0, 75, 300, or 1200 ppm); male NBR rats were exposed for 2 weeks. The kidney and the liver were targets of PGMBE toxicity in rats. Renal lesions suggestive of alpha(2u)-globulin nephropathy were observed in male F344/N, in the 2 and 14-week studies, no kidney lesions were seen in NBR rats. In the 2-year study, male rats displayed exposure-related nonneoplastic lesions in the kidney, and may have shown marginal increases in tubular neoplasms. In the liver, the incidences of hepatocellular adenomas occurred with a positive trend in male rats, and may have been related to PGMBE exposure. In mice of both sexes, the major target of PGMBE toxicity was the liver. In the 2-week study, liver weights and in the 14-week study, liver weights and the incidences of centrilobular hypertrophy were increased. In the 2-year study, the incidences of exposure-related hepatocellular adenoma, adenoma or carcinoma combined, and hepatoblastoma occurred with a positive trend, and were significantly increased in 1200 ppm groups. In summary, exposure to PGMBE resulted in nonneoplastic lesions of the kidney characteristic of alpha(2u)-globulin nephropathy, and may have increased renal tubular neoplasms in male rats. Exposure to PGMBE also produced increases in hepatic tumors in male and female mice.

MeSH terms

  • Adenoma, Liver Cell / chemically induced
  • Adenoma, Liver Cell / pathology
  • Administration, Inhalation
  • Alpha-Globulins / metabolism
  • Animals
  • Carcinogenicity Tests
  • Carcinogens / administration & dosage
  • Carcinogens / toxicity*
  • Carcinoma, Hepatocellular / chemically induced
  • Carcinoma, Hepatocellular / pathology
  • Dose-Response Relationship, Drug
  • Female
  • Hepatoblastoma / chemically induced
  • Hepatoblastoma / pathology
  • Kidney Neoplasms / chemically induced
  • Kidney Neoplasms / pathology
  • Kidney Tubules / drug effects
  • Kidney Tubules / metabolism
  • Kidney Tubules / pathology
  • Liver Neoplasms / chemically induced
  • Liver Neoplasms / pathology
  • Male
  • Mice
  • Mice, Inbred Strains
  • Mutagens / toxicity
  • Propylene Glycols / administration & dosage
  • Propylene Glycols / toxicity*
  • Rats
  • Rats, Inbred F344
  • Salmonella typhimurium / drug effects
  • Salmonella typhimurium / genetics
  • Solvents / administration & dosage
  • Solvents / toxicity*

Substances

  • Alpha-Globulins
  • Carcinogens
  • Mutagens
  • Propylene Glycols
  • Solvents
  • alpha 2u globulin
  • propylene glycol mono-t-butyl ether