The pathology of ductal-type pancreatic carcinomas and pancreatic intraepithelial neoplasia: insights for clinicians

Curr Gastroenterol Rep. 2004 Apr;6(2):111-8. doi: 10.1007/s11894-004-0037-y.

Abstract

The phenotypic classification of pancreatic neoplasms is based on their cellular lineage. Thus, tumors with a ductal, acinar, and endocrine phenotype can be distinguished. Most pancreatic neoplasms show a ductal phenotype and can be classified as ductal adenocarcinomas. Less common tumors with a ductal phenotype are the variants of ductal adenocarcinoma, intraductal papillary mucinous neoplasm (including colloid carcinoma), mucinous cystic neoplasm, medullary carcinoma, and other rare tumors. Ductal adenocarcinomas most likely develop from ductal proliferative lesions arising in the pancreatic duct system. A recently adopted classification system for these lesions distinguishes between three grades of pancreatic intraepithelial neoplasia (PanIN). Molecular studies have revealed that PanIN-2 and PanIN-3 lesions represent a distinct step toward invasive carcinoma.

Publication types

  • Review

MeSH terms

  • Adenocarcinoma / pathology*
  • Adenocarcinoma, Mucinous / pathology
  • Carcinoma in Situ / pathology*
  • Carcinoma, Pancreatic Ductal / pathology*
  • Disease Progression
  • Humans
  • Mucin-2
  • Mucins / genetics
  • Neoplasm Proteins / genetics
  • Oligonucleotide Array Sequence Analysis
  • Pancreatic Neoplasms / genetics
  • Pancreatic Neoplasms / pathology*

Substances

  • MUC2 protein, human
  • Mucin-2
  • Mucins
  • Neoplasm Proteins